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Fibrinogen and LDL Apheresis in Treatment of Sudden Hearing Loss: A Randomized Multicentre Trial

The Lancet
2002;360:1811-1817.

M Suckfüll, for the Hearing Loss Study Group

The causes of sudden sensorineural hearing loss (SSHL) are unclear but are believed to include changes in microcirculation, autoimmune pathology, or viral infection. The identification of various circulatory disturbances in some individuals suffering from SSHL supports the role of vascular pathogenesis in this condition. A pilot study of SSHL in patients with hyperfibrinogenemia or hypercholesterolemia found that LDL-apheresis was more successful in restoring hearing than was the standard therapy with steroids, dextrane, and pentoxifylline.* It is suggested that the decreased levels of plasma fibrinogen and the increased release of nitric oxide due to reduced serum cholesterol following LDL-apheresis result in improved blood flow.

The present study investigated whether a single fibrinogen/LDL-apheresis treatment was equally or more effective than a standard treatment of prednisolone, hydroxyethyl starch, and pentoxifylline in treating SSHL. A total of 201 patients were enrolled, of which 134 were randomly assigned to the apheresis group and 67 to the standard-treatment group. The primary outcome was recovery of hearing as measured by pure-tone audiometry 48 hours following the start of treatment and was assessed in 111 (83%) apheresis-group patients and in 59 (88%) standard-group patients. Secondary outcomes were the recovery of hearing 6 weeks posttreatment, improvement of speech audiometry, tinnitus, and frequency of side effects. Secondary outcomes were assessed for 109 and 54 patients in the apheresis and standard groups, respectively.

Hearing improved in both groups at 48 hours and 6 weeks posttreatment, but with no significant differences between the groups. In contrast, a significant difference (P=0.034) in mean speech perception was evident 48 hours posttreatment in the apheresis group (21.6 ± 20.8 decibels [dB]) compared with the standard group (29.3 ± 29.4 dB). This difference was still detectable at 6 weeks (13.7 ± 14.3 dB vs 20.8 ± 25.4 dB, P= 0.059) but was not deemed significant.

Subgroup analysis demonstrated that patients with median plasma fibrinogen concentrations above 8.68 µmol/L showed greater improvement in speech perception following apheresis (15.3 ± 2.5 dB) compared with treatment by standard means (6.1 ± 2.0 dB, P=0.005). Similar findings were seen in patients with LDL-C levels above 3.47 mmol/L (11.2 ± 1.7 dB vs 4.6 ± 1.6 dB).

The authors conclude that a single fibrinogen/LDL-apheresis treatment can be used as an alternative to conventional treatments with infusions and steroids for 10 days.

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* See also: Suckfüll M, Thiery J, Schorn K, Kastenbauer E, Seidel D. Clinical utility of LDL-apheresis in the treatment of sudden hearing loss: a prospective randomized study. Acta Otolaryngol. 1999;119:763-766.



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